The Mitochondrial Permeability Transition Pore – a Regulator of the Resistance of the Heart to Reperfusion; Neuroscience and Behavioral Physiology; Vol. 48, iss. 8

Manylion Llyfryddiaeth
Parent link:Neuroscience and Behavioral Physiology.— , 1967-
Vol. 48, iss. 8.— 2019.— [P. 943–953]
Awdur Corfforaethol: Национальный исследовательский Томский политехнический университет (ТПУ) Физико-технический институт (ФТИ) Лаборатория № 31 ядерного реактора (Лаборатория № 31 ЯР)
Awduron Eraill: Naryzhnaya N. V. Nataliya Vladimirovna, Maslov L. N. Leonid Nikolaevich, Lishmanov Yu. B. Yury Borisovich, Nesterov E. A. Evgeny Alexandrovich, Jaggi A. S. Amteshwar Singh, Suleiman S. Saadeh
Crynodeb:Title screen
The most important event in the formation of reperfusion injury to the heart has been shown to be the opening of mitochondrial permeability transition pores (mPTP). mPTP opening is followed by activation of apoptosis, necroptosis, and cardiomyocyte death. Experimental studies have shown that inhibition of mPTP opening decreases infarct size and that conditioning actions on the myocardium are mediated via decreases in the sensitivity of mPTP to opening factors. The present article reviews existing data on the structure and regulation of mPTP in conditions of reperfusion injury to the heart.
Режим доступа: по договору с организацией-держателем ресурса
Iaith:Saesneg
Cyhoeddwyd: 2019
Pynciau:
Mynediad Ar-lein:https://doi.org/10.1007/s11055-019-00823-y
Fformat: MixedMaterials Electronig Pennod Llyfr
KOHA link:https://koha.lib.tpu.ru/cgi-bin/koha/opac-detail.pl?biblionumber=663726

MARC

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200 1 |a The Mitochondrial Permeability Transition Pore – a Regulator of the Resistance of the Heart to Reperfusion  |f N. V. Naryzhnaya, L. N. Maslov, Yu. B. Lishmanov [et al.] 
203 |a Text  |c electronic 
300 |a Title screen 
320 |a [References: 130 tit.] 
330 |a The most important event in the formation of reperfusion injury to the heart has been shown to be the opening of mitochondrial permeability transition pores (mPTP). mPTP opening is followed by activation of apoptosis, necroptosis, and cardiomyocyte death. Experimental studies have shown that inhibition of mPTP opening decreases infarct size and that conditioning actions on the myocardium are mediated via decreases in the sensitivity of mPTP to opening factors. The present article reviews existing data on the structure and regulation of mPTP in conditions of reperfusion injury to the heart. 
333 |a Режим доступа: по договору с организацией-держателем ресурса 
461 |t Neuroscience and Behavioral Physiology  |d 1967- 
463 |t Vol. 48, iss. 8  |v [P. 943–953]  |d 2019 
610 1 |a электронный ресурс 
610 1 |a труды учёных ТПУ 
610 1 |a myocardium 
610 1 |a reperfusion 
610 1 |a MPT pores 
610 1 |a mitochondria 
610 1 |a миокард 
610 1 |a реперфузия 
610 1 |a митохондрии 
701 1 |a Naryzhnaya  |b N. V.  |g Nataliya Vladimirovna 
701 1 |a Maslov  |b L. N.  |g Leonid Nikolaevich 
701 1 |a Lishmanov  |b Yu. B.  |c specialist in the field of medical technology  |c lead engineer aof Tomsk Polytechnic University, doctor of medical sciences  |f 1951-  |g Yury Borisovich  |3 (RuTPU)RU\TPU\pers\34200 
701 1 |a Nesterov  |b E. A.  |c Physicist, Specialist in the field of nuclear power engineering  |c Researcher of Tomsk Polytechnic University  |f 1976-  |g Evgeny Alexandrovich  |3 (RuTPU)RU\TPU\pers\32657  |9 16556 
701 1 |a Jaggi  |b A. S.  |g Amteshwar Singh 
701 1 |a Suleiman  |b S.  |g Saadeh 
712 0 2 |a Национальный исследовательский Томский политехнический университет (ТПУ)  |b Физико-технический институт (ФТИ)  |b Лаборатория № 31 ядерного реактора (Лаборатория № 31 ЯР)  |3 (RuTPU)RU\TPU\col\20054 
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