Phototoxicity of flavoprotein miniSOG induced by bioluminescence resonance energy transfer in genetically encoded system NanoLuc-miniSOG is comparable with its LED-excited phototoxicity; Journal of Photochemistry and Photobiology B: Biology; Vol. 188

গ্রন্থ-পঞ্জীর বিবরন
Parent link:Journal of Photochemistry and Photobiology B: Biology
Vol. 188.— 2018.— [P. 107-115]
সংস্থা লেখক: Национальный исследовательский Томский политехнический университет (ТПУ) Физико-технический институт (ФТИ) Лаборатория радиационного контроля № 31 (Лаборатория РК № 31)
অন্যান্য লেখক: Proshkina G. M. Galina Mikhaylovna, Shramova E. I. Elena Ivanovna, Shilova O. N. Olga Nikolaevna, Ryabova A. V. Anastasiya Vladimirovna, Deev S. M. Sergey Mikhaylovich
সংক্ষিপ্ত:Title screen
Photodynamic therapy (PDT) is a clinical, minimally invasive method for destroying cancer cells in the presence of a photosensitizer, oxygen, and a light source. The main obstacle for the PDT treatment of deep tumors is a strong reduction of the excitation light intensity as a result of its refraction, reflection, and absorption by biological tissues. Internal light sources based on bioluminescence resonance energy transfer can be a solution of this problem. Here we show that luciferase NanoLuc being expressed as a fusion protein with phototoxic flavoprotein miniSOG in cancer cells in the presence of furimazine (highly specific NanoLuc substrate) induces a photodynamic effect of miniSOG comparable with its LED-excited (Light Emitting Diode) phototoxicity. Luminescence systems based on furimazine and hybrid protein NanoLuc-miniSOG targeted to mitochondria or cellular membranes possess the similar energy transfer efficiencies and similar BRET-induced cytotoxic effects on cancer cells, though the mechanisms of BRET-induced cell death are different. As the main components of the proposed system for BRET-mediated PDT are genetically encoded (luciferase and phototoxic protein), this system can potentially be delivered to any site in the organism and thus may be considered as a promising approach for simultaneous delivery of light source and photosensitizer in deep-lying tumors and metastasis anywhere in the body.
Режим доступа: по договору с организацией-держателем ресурса
ভাষা:ইংরেজি
প্রকাশিত: 2018
বিষয়গুলি:
অনলাইন ব্যবহার করুন:https://doi.org/10.1016/j.jphotobiol.2018.09.006
বিন্যাস: MixedMaterials বৈদ্যুতিক গ্রন্থের অধ্যায়
KOHA link:https://koha.lib.tpu.ru/cgi-bin/koha/opac-detail.pl?biblionumber=658876

MARC

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200 1 |a Phototoxicity of flavoprotein miniSOG induced by bioluminescence resonance energy transfer in genetically encoded system NanoLuc-miniSOG is comparable with its LED-excited phototoxicity  |f G. M. Proshkina [et al.] 
203 |a Text  |c electronic 
300 |a Title screen 
320 |a [References: p. 114-115 (54 tit.)] 
330 |a Photodynamic therapy (PDT) is a clinical, minimally invasive method for destroying cancer cells in the presence of a photosensitizer, oxygen, and a light source. The main obstacle for the PDT treatment of deep tumors is a strong reduction of the excitation light intensity as a result of its refraction, reflection, and absorption by biological tissues. Internal light sources based on bioluminescence resonance energy transfer can be a solution of this problem. Here we show that luciferase NanoLuc being expressed as a fusion protein with phototoxic flavoprotein miniSOG in cancer cells in the presence of furimazine (highly specific NanoLuc substrate) induces a photodynamic effect of miniSOG comparable with its LED-excited (Light Emitting Diode) phototoxicity. Luminescence systems based on furimazine and hybrid protein NanoLuc-miniSOG targeted to mitochondria or cellular membranes possess the similar energy transfer efficiencies and similar BRET-induced cytotoxic effects on cancer cells, though the mechanisms of BRET-induced cell death are different. As the main components of the proposed system for BRET-mediated PDT are genetically encoded (luciferase and phototoxic protein), this system can potentially be delivered to any site in the organism and thus may be considered as a promising approach for simultaneous delivery of light source and photosensitizer in deep-lying tumors and metastasis anywhere in the body. 
333 |a Режим доступа: по договору с организацией-держателем ресурса 
461 1 |t Journal of Photochemistry and Photobiology B: Biology 
463 1 |t Vol. 188  |v [P. 107-115]  |d 2018 
610 1 |a электронный ресурс 
610 1 |a труды учёных ТПУ 
610 1 |a miniSOG 
610 1 |a Bioluminescence resonance energy transfer (BRET) 
610 1 |a BRET-mediated phototoxicity 
610 1 |a BRET efficiency 
610 1 |a NanoLuc 
610 1 |a передача энергии 
610 1 |a резонанс 
610 1 |a биолюминесценция 
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701 1 |a Shramova  |b E. I.  |g Elena Ivanovna 
701 1 |a Shilova  |b O. N.  |g Olga Nikolaevna 
701 1 |a Ryabova  |b A. V.  |g Anastasiya Vladimirovna 
701 1 |a Deev  |b S. M.  |c biologist  |c the expert of Tomsk Polytechnic University, doctor of biological Sciences  |g Sergey Mikhaylovich  |3 (RuTPU)RU\TPU\pers\39299 
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