The Question of the End Effector of Ischemic Preconditioning of the Heart; Neuroscience and Behavioral Physiology; Vol. 47, iss. 6
| Parent link: | Neuroscience and Behavioral Physiology.— , 1967- Vol. 47, iss. 6.— 2017.— [P. 681–689] |
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| Autor corporatiu: | , |
| Altres autors: | , , , , , , |
| Sumari: | Title screen The authors' analysis of literature sources provided evidence that the main candidates for the role of end effector of ischemic preconditioning of the heart are: MPT pores (mitochondrial permeability transition pores), nexuses, and the cytoskeleton. Almost all known intracellular signal cascades ultimately converge on MPT pores, cytoskeletal components, and nexuses. Режим доступа: по договору с организацией-держателем ресурса |
| Idioma: | anglès |
| Publicat: |
2017
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| Matèries: | |
| Accés en línia: | http://dx.doi.org/10.1007/s11055-017-0455-6 |
| Format: | Electrònic Capítol de llibre |
| KOHA link: | https://koha.lib.tpu.ru/cgi-bin/koha/opac-detail.pl?biblionumber=657961 |
| Sumari: | Title screen The authors' analysis of literature sources provided evidence that the main candidates for the role of end effector of ischemic preconditioning of the heart are: MPT pores (mitochondrial permeability transition pores), nexuses, and the cytoskeleton. Almost all known intracellular signal cascades ultimately converge on MPT pores, cytoskeletal components, and nexuses. Режим доступа: по договору с организацией-держателем ресурса |
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| DOI: | 10.1007/s11055-017-0455-6 |