Association Study Indicates a Protective Role of Phosphatidylinositol-4-Phosphate-5-Kinase against Tardive Dyskinesia; International Journal of Neuropsychopharmacology (IJNP); Vol. 18, iss. 6

Λεπτομέρειες βιβλιογραφικής εγγραφής
Parent link:International Journal of Neuropsychopharmacology (IJNP)
Vol. 18, iss. 6.— 2015.— [6 р.]
Συγγραφή απο Οργανισμό/Αρχή: Национальный исследовательский Томский политехнический университет Институт неразрушающего контроля Кафедра экологии и безопасности жизнедеятельности
Άλλοι συγγραφείς: Fedorenko O. Yu. Olga Yurievna, Lunen A. D. Anton DzhM, Florian L. Lang, Toshchakova V. A. Valentina Aleksandrovna, Boyarko E. G. Evgeniya Grigorjevna, Semke A. V. Arkady Valentinovich, Bokhan N. A. Nikolay Aleksandrovich, Govorin N. V., Aftanas L. V. Lyubomir Ivanovich, Ivanova S. A. Svetlana Aleksandrovna
Περίληψη:Title screen
Tardive dyskinesia is a disorder characterized by involuntary muscle movements that occur as a complication of long-term treatment with antipsychotic drugs. It has been suggested to be related to a malfunctioning of the indirect pathway of the motor part of the cortical-striatal-thalamic-cortical circuit, which may be caused by oxidative stress-induced neurotoxicity.
Γλώσσα:Αγγλικά
Έκδοση: 2015
Θέματα:
Διαθέσιμο Online:http://dx.doi.org/10.1093/ijnp/pyu098
Μορφή: Ηλεκτρονική πηγή Κεφάλαιο βιβλίου
KOHA link:https://koha.lib.tpu.ru/cgi-bin/koha/opac-detail.pl?biblionumber=648927
Περιγραφή
Περίληψη:Title screen
Tardive dyskinesia is a disorder characterized by involuntary muscle movements that occur as a complication of long-term treatment with antipsychotic drugs. It has been suggested to be related to a malfunctioning of the indirect pathway of the motor part of the cortical-striatal-thalamic-cortical circuit, which may be caused by oxidative stress-induced neurotoxicity.
DOI:10.1093/ijnp/pyu098